Rip1 and PdtaR mutant transcriptional responses to Nitric oxide

Description

BioProject Summary: "Analysis of the transcriptional response of M. tuberculosis Erdman lacking the Rip1 protease, the PdtaR response regulator, or both, to the NO donor DETA-NO" Abstract summary: "Bacterial pathogens that infect phagocytic cells must deploy mechanisms that sense and neutralize host microbicidal effectors. For Mycobacterium tuberculosis, the causative agent of tuberculosis, these...

Subject

Mycobacterium tuberculosis

Triazenes

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